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The effects of MET activation are crucial to physiological processes such as embryonic development, organ development and wound healing [7]

Thus, we propose QMP engineering as a strategy to ensure a long plasma half-life and enhanced therapeutic efficacy of albumin fusion proteins despite C-terminal cleavage

It does not establish a validated human dose, half-life, or dosing schedule for either compound

To avoid muscle mass by GLP-1 analogs, combination therapy of muscle mass inducer such as Bimagrumab (Anti-ACVR2B Antibody) [54] with GLP-1 analogs would be a good treatment regimen to suppress muscle mass reduction leads to sarcopenia