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Pathways of mitochondrial transfer The intercellular transfer of mitochondria, a crucial compensatory mechanism in pathological conditions such as IS [17, 18], is mediated through multiple highly coordinated pathways including [1] tunneling nanotubes (TNTs) [2], extracellular vesicles (EVs) [3], connexin 43 (Cx43)-mediated gap junctions, and [4] direct membrane fusion processes [19, 20]

Malondialdehyde-acetaldehyde adduct is the dominant epitope after MDA modification of proteins in atherosclerosis

Conversely, in neuroinflammatory diseases, astrocytes are excessively activated into a neurotoxic state by cytokines secreted by microglia, releasing uncontrolled pro-inflammatory cytokines and complement proteins, which exacerbate damage to neighboring cells (6,7)

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