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This landmark study demonstrates that Parkin selectively translocates to depolarized mitochondria to promote their autophagic clearance, establishing mitophagy as a quality-control pathway for damaged mitochondria

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Because IGF1/IGF1r is closely linked to the mTOR pathway, and mTOR is a crucial negative regulator of autophagy, We hypothesize that IGF1 exacerbates autophagic dysfunction through the activation of IGF1r in the TNC brain region, leading to neuronal hyperactivation and the manifestation of headache behaviors