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Our analyses displayed that endometriosis could be polyepigenetic and with alterations in specific genes implicated in major signaling pathways contributing to the disease etiopathology (cell proliferation, differentiation, and division [PI3K-Akt and Wnt-signaling pathway], cell division [MAPK pathway], cell adhesion, cell communication, developmental processes, response to hormone, apoptosis, immunity, neurogenesis, and cancer)

Multiple immune-related adverse events and anti-tumor efficacy: real-world data on various solid tumors

During development, CHAC2 exhibits a role complementary to CHAC1 by enhancing pluripotency and stem cell renewal, whereas CHAC1 promotes differentiation

While there has been much research into the mechanisms of damage related to mycotoxins, the link between metabolism, toxicity and carcinogenicity of mycotoxins remains incompletely understood [10]