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Moreover, increased pPERK immunoreactivity colocalizes with -synuclein, suggesting the loss of proteostasis and the activation of the UPR in degenerating neurons (Hoozemans et al., 2007)

Similar results were seen in a Mayo clinic study where a high prevalence of nasal fungal growth was found in both symptomatic rhinosinusitis patients and controls [97] with the most significant difference being the development of eosinophilic mucin and not type 1 hypersensitivity since IgE positivity was not seen in the majority of patients [97]

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