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Type-2 astrocyte-like cells are more resistant than oligodendrocyte-like cells against non- N -methyl-D-aspartate glutamate receptor-mediated excitotoxicity

132 Overall, the fibrosis-promoting and fibrosis-suppressing functions of senescence seemed to be incompatible, but in fact most senescent cells secrete SASPs that impact fibrosis development, which is a vital mechanism in OSF progression and should be comprehensively explored in the future

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This novel treatment strategy holds promise in enhancing patient outcomes by effectively managing the severe inflammatory responses associated with COVID-19 [94]